Hair Transplant Gone Wrong

I have discussed hair transplants on this blog many times in the past. Modern day hair restoration procedures usually provide very satisfying results when performed by reputable accredited hair transplant surgeons. Major skin related permanent side effects or severe health problems during and after a hair transplant procedure are becoming a rarity.

However, even the best surgeons out there will have some bad results due to unforeseen circumstances or, in rare cases, the surgeon or assisting technician having a bad day. Nevertheless, this risk is much smaller in countries such as the US where official accreditation is indicative of at least a minimum level of competency.

On the other hand, if you get a hair transplant abroad in order to save money, you will need do be very careful in selecting your surgeon. You want to make sure the surgeon is accredited by the non-profit ISHRS, is locally board certified, has decent online reviews and allows you to meet a few of his or her past patients.

Earlier today, this issue was brought forward front and center into my mind after Pakistan based actor Sajid Hasan posted a graphic video online about his botched hair transplant gone wrong. It seems like the person who did this procedure several weeks ago might have been a medical doctor or general surgeon, and not even an official hair transplant surgeon.

Besides having persistent health related side effects, Mr. Hasan’s scalp currently looks like something that was worked upon by a butcher:

Hair Transplant Gone Wrong
Hair Transplant Gone Wrong

 

112 thoughts on “Hair Transplant Gone Wrong”

  1. There is another website/radio show that only ever seem to talk about FUE, FUT, etc on and on and on. (You know the one). Maybe they should take a look at this one?! I feel sick. Poor guy. I’ve seen a hair transplant patient mid procedure. It ain’t pretty. I personally have never seen a hair transplant up close that didn’t look kinda odd?! I’m sure they exist but I don’t think I would ever risk it-nor could I afford it. One scumbag hairloss doc was pushing me to get one even though I don’t have stabalized hairloss-so I would never be a good candidate anyway. He said it would last “at least” two years…yeah great. Got outta there real quick.

  2. Hair transplants are so tempting. It’s like, do I get one now and risk that Follica/Aclaris have a tremendous treatment out right after I get one, or do I wait? I’ve decided just to wait and accept my hairloss for now. I’d love a thick head of hair, but it just isn’t in the cards genetically. I’d rather look naturally bald than unnaturally restored. As the commenter above wrote, you can always sorta tell when something isn’t natural. I feel that looking unnatural is worse than looking like you had surgery; esp for men.

    I also think about famous men like Tim Allen and Kenny Rogers; have you seen their eyes?!? They both have had stuff done and look feminized and unnatural.

    1. Hey Daniel,

      I also have very bad genetics which probably don’t make a strong ht candidate either however i have seen good results from ht + non permanent smp if done by the right people. Preferable if very short hair suits you. Other than that i would agree that it’s better not to get anything done otherwise looks unnatural.

      1. I’ve heard of smp and some of the results I’ve seen are good. I like the idea of keeping hair short anyway so maybe it’d be a decent choice. Like you said though, I definitely would only consider temporary pigmentation to keep my future options open. No clue how much it costs either.

  3. Phew, I thought I missed a Polarity post from 2016 I was about to flip out lol. Yeah great find admin seriously.

    Mjones, I’m no god at investing. markets make anyone over time a humble cynic. I want to buy COOL too but I’ve got my money tied up into a statistical arbitrage algo my brother programmed. If we could get some time line news release quotes of market release or something to run on it would be trade-able .. Only problem is SP500 had a huge correction it’s a risk off environment so it might be a sketchy time to get in.

    This site informed me of CRISPR and I almost doubled a trade on EDIT’s big move 2016 only difference is if Polarity is the real deal it’s going to be way bigger way sooner. but i’m still skeptical we need a time line of a news release like if they say “we plan a release of whateverTE on x date/x quarter/x month” it’s better to get in that time frame rather than right now while the SP500 is collapsing, trading alongside macro rather than against it.

    I vaguely remember them saying something about pig test being successfully completed moving into humans first quarter and they are already registered with the FDA .. I don’t know anything else at the moment.

  4. I had a hair transplant 3 years ago in Harley Street, London. The most advanced top medical treatments in the UK are done there, from cosmetic stuff to everything else. Expensive but I did not want to take any risks.
    Credit to the surgeon, he did what he could and I have had no adverse side effects, but my hair was thin and I had already lost a lot so I did not get the result I wanted. I wish the surgery could have made more of an impact but that is not their fault.

    If I knew what I know now about the promising new treatments I would not have bothered and I advise you all to do the same. Wait 2 years for all these players to either release the cure or drop out of the game, then make a decision then; because if Aclaris, Shiseido, Follicum and Tsuji all fail then what the hell is there left to try? :-)

      1. @Netshed I gave up with it in the end and shaved it all off, in protest until a cure is released that makes it full. It’s better to look like “I don’t care” rather than “I am insecure” even though the latter is true its all about the weaknesses we choose to show. To my surprise I get more attention from females being shaven, which would otherwise be good but I am gay and my last 3 boyfriends preferred good hair. Never mind :-)

          1. I had a FUT that was very visible for the first 6 months and FUE that was visible for 3. But after a year I can shave my head and see nothing. If that is all you are worried about then no worries, but I would wait for the big players in the next couple of years to either deliver or fold, then consider your options.

            1. Great advice Scott thank you for sharing your story with us.

              I’ll pretty much do anything to avoid a transplant no matter how tempting it is (and it is). I’m sitting here right now with a laser helmet on my head I’ve gone that far haha

          1. Absolutely rubbish. Everyone has heard of harley street in London. Turkey is absolutely terrible at making anything look natural. You get what you pay for.

            1. Yes, everyone knows harley street london, but that wasn’t my argument. It is not known for decent hair transplant surgeons that perform mega sessions. Search around the internet forums.

              Just accept it, you chose a terrible location to have your surgery . Erdogan in Turkey is a pioneer in the FUE technique, and if you do your research you will discover he is a vice president of the World FUE institute:

              https://en.wikipedia.org/wiki/World_FUE_Institute

              Doctors in Harley street do not know how to perform large scale sessions, their knowledge is outdated as far as FUE goes.

            1. In 2009 I flew from the UK to Canada for Dr Rahal. I have very fine hair. He did a great job that has stood the test of time. I’d opt for him again if going for another procedure rather than a Harley street surgeon. N America has the best guys IMO.

    1. Scott, how come you can be so optimistic about future treatments? None of this companies provided any results, that you could be promising. And very long way to being finally tested and released. Pleace explain.

      I wish to have your optimism… I just found out that fevi is moved to 2020… Great :/

      1. @ffar, considering that a baldness cure has been searched for since before the Pyramids were built, the fact that it will happen in your lifetime at all is something of a miracle. If it is 2030 that would still be great. Hair is just cosmetic and I can do all the stuff I want to do in mjy life without it; women, men, drugs, parties, sport, xbox, studying, vacations abroad. You don’t get stopped at airport customs for being bald :-) 2020 is not that far away and the Shiseido phase 2 trial ends late this year, plus that Aclaris trial that the other users on here get excited about but I don’t rate.
        It’s all good my friend.

  5. Not sure if this is new or not but the Puretech Pipeline website ( http://puretechhealth.com/our-pipeline/wound-induced-neogenesis-rain ) appears to have some minor adjustments. They are calling it “Abrasion Induced Neogenesis”. So it sounds like RAIN is an acronym for Regenerative Abrasion Induced Neogenesis. They still seem to be on target for their 2018 trial although it looks like there will be 2 trials, a pre-pivotal and pivotal, one on top of the other: “Our regenerative biology program is currently progressing into a pre-pivotal pilot optimization study expected to start in early 2018, with a pivotal trial expected to begin in the first half of 2018”

      1. Do you recall if the “pre-pivotal” trial is a new addition? I am wondering if this is a very small quick test before they sink their money into the larger trial or perhaps they are trying alternate compounds as a result of continuing research and they will test more than one version in their larger trial if the pre-pivotal compound shows promise.

        1. I don’t remember reading about any pre-pivotal trial before. Am at a work related job right now, but will check in more detail later today.

      1. If it creates follicles de novo, I don’t see why you couldn’t repeat after allowing time for the scalp to heal and regenerate. On the surface, I think this sounds like a reasonable possibility. On another note, I know Follica had explored the possibility of disrupting the scalp without having to shave the head. I wonder if this means they have decided otherwise.

        1. The patents for the in-office device specifically state that it has an attachment that allows it to bypass existing hair.

      1. I remember reading that patent before but didn’t know if they could actually make the device work………..do you have a link to that patent? In re-reading some of the patents, I do see where they suggest that multiple treatments may “build up hair density over time”………….and that “serial perturbations” may “produce an additive hair growth effect” so Drew wasn’t really dreaming.

    1. I’ve been using a very stiff brush aggressively on my scalp last 3 months, 2-3 mins twice a day. It’s increased hair density much more than any other treatment I’ve tried.

    1. I just heard about this on the local news ! Turns out its tied to one of last articles the admin posted about hair follicle germs ! Lots of players out their as of late ! I think were getting close .

  6. I had a fue 4 years ago, 2500 grafs with a top doctor, I’d choose a conservative hair line, it looks natural. Nobody would tell…if you didn’t know me before, because I look like another person and of course feel confident and happy. I would never do it with a cheap doctor if I couldn’t pay for a top, even in that case always is a risk.

    1. Hands down to China for prosecuting those scam artists. I wish more countries would Crack down on these con artists in the hair loss world. Plus Crack down on unethical ht surgeons that ruin peoples lives.

    1. well there was a small announcement with timelines i was babbling about and there was a 7% lift in stock then back down again the same day because sp500 lost 5% in one day

  7. Fukuda looks like the real deal but as always 10 years away. Plus we shouldn’t get too optimistic until they try it on humans in 5 years lol. The positive side is they are still looking for new ways to clone. How is this different than tsuji?

    Have you guys heard of Fue Experts out of Madrid spain? There Fue results look freaking amazing! They have a wait list till 2022. I heard someone say that they secretly are performing hair cloning there. Either way that ht doc is a freaking pro! Great work if legit.

        1. I spoke with both.

          dr Couto do not take any consultation at the moment… And waiting time for people who had consultation booked for “today” have to wait 3,5 years for procedure.

          dr Freitas has waiting time for consultation 1,5 year(I have mine in April 2019). Waiting time for operation after will be over a year.

          Both of them as far as I know don’t operate without fin :/ I am really hope that before that I will be able to get Fevi.

          1. HI Dean…would he accept people who are losing effects of fin. His results look really really good! Did he mention how they get such superior results compared to other surgeons in the USA and Europe Turkey?

            1. Don’t know. But that’s good question. He seams to believe that fin is stabilize hair loss, and that’s why he require to use it.

              I was refused by dr Erdogan to have HT, because he said i don’t have hair loss but miniaturization… With i don’t think is a case, but whatever…

              I am not willing to take FIN, seti/fevi yes, but not FIN. But best would be RCH-01… It is devastating to wait for report from shiseido.

  8. Admin, maybe you have a good answer for this:

    We are able to grow hair on mice. The main issue has been controlling the direction of hair growth and the concern of uncontrolled proliferation leading to cancerous growth. These can be eliminated by having a medium such as lab grown skin or synthetic skin, which we already have. Why can’t we implant the cells into it, let it grow then transplant it to the human. Or better yet, (PETA please stop reading) we can use immunosuppressed mice that can host these cells to growth then transplant it from the mice to the human until we figure out a better way. We essentially have the cure at this point.

  9. have u seen trumps hair? I always thought of an idea how ur hair would look like if you deplete all donor cells and transplant to the top.. now I know how it looks

  10. I’m no fan of Trumpy, but I did feel a modicum of pity for him getting on that plane. It reminded me of a scene in The Empire Strikes Back where Darth Vader is sitting in his chamber and you get a glimpse of his badly scared head. I’m not comparing him to Darth Vader or anything, no, no, no…Vader is far too nice.

    1. looling like Samumed is still working on Aga. They finally mentioned Aga in a twitter feed. Starting to think it was over but nope. Next week in San Diego they have a booth set up at an event, to answer questions on Aga and whatever else.

      1. Please Check comments of Maxim Jacobs, CFA, Director of Healthcare Research, North America for Edison Investment Research, on samumed.

        He mentioned, “The greater fool theory has become the basis for valuation” with related to data on their developments regarding hair.

  11. From Samumed Twitter feed:
    Samumed
    @Samumed_LLC
    16h
    Going to #AAD18 next week? Visit us at booth #5053 to hear about potential treatment options in development for patients suffering with #psoriasis and #androgeneticalopecia.

    1. “AGA is the result of chronic GA-transmitted scalp tension mediated by pubertal and post-pubertal skull bone growth and/or the overdevelopment and chronic contraction of muscles connected to the GA. This tension induces a pro-inflammatory cascade (increased ROS, COX-2 signaling, IL-1, TNF-α, etc.) which induces TGF-β1 alongside increased androgen activity (5-αR2, DHT, and AR), which furthers TGF-β1 expression in already-inflamed AGA-prone tissues. The concomitant presence of DHT and TGF-β1 mediates perifollicular fibrosis, dermal sheath thickening, and calcification of the capillary networks supporting AGA-prone hair follicles. These chronic, progressive conditions are the rate-limiting factors in AGA recovery. They restrict follicle growth space and decrease oxygen and nutrient supply to AGA-prone tissues – leading to tissue degradation, hair follicle miniaturization, and eventually pattern baldness.”

      1. “This model allows for genetic influence during any step-process, but refutes the belief that AGA-prone follicles are genetically programmed to become sensitive to DHT. Rather, the model implies that AGA-prone tissues are predisposed to respond to chronic tension-mediated inflammation by inducing DHT and androgen-mediated TGF-β1, which restructure tissue – of which a symptom is hair loss. The model also provides a rationale for unexplained phenomena in AGA pathology, such as:

        •Why DHT increases in AGA-prone scalp tissues (i.e., DHT is a response to tension-mediated inflammation)
        •The mechanisms by which DHT is involved in AGA progression (i.e., DHT is involved in the onset of fibrosis and calcification)
        •The pattern of AGA (i.e., AGA progression matches that of where GA-transmitted scalp tension is highest, and progresses as peak tension points change during fibrosis onset)
        •Why AGA is observed more often in elderly populations versus young adults (i.e., calcification and fibrosis have had more time to accumulate)
        •Why DHT is associated with body and facial hair growth and also AGA-related hair loss (i.e., tension-mediated inflammation induces TGF-β1 and DHT, and remodels tissue in AGA sites – a phenomenon not observed in body and facial hair growth sites)
        •Why androgen suppression stops AGA, but does not regrow all hair (i.e.; DHT inhibitors may reduce fibrosis progression in AGA, but do not reverse fibrosis already present).”

        1. “Future AGA research should focus on utilizing mechanotransduction to potentially reverse AGA-related tissue remodeling. If the model holds true, then reversing AGA tissue remodeling – rather than attenuating it – may pave the pathway to full AGA recoveries.”

          1. @OMG
            posted this article earlier in the comments beneath the Poietis-3d 4d post, however yesterday I had time to find and read the entire pdf. I think it’s a must-read.

            It also clarifies why FIN and MIN (at the end) can’t win the battle. Only slow it down if we are lucky.

    2. Good stuff netshed. I do daily scalp massages and I don’t get that burning or crawling feeling on my scalp like I did a few years ago. Plus my dandruff has gone down as well. I’m still losing ground but I don’t have that pain anymore. Inflammation is def the main culprit followed by fibrosis. Jak will tackle that angle. Hypothetically Jak topical with finasteride combo should do the trick. Or tsuji comes out and cures us all and we get move on from this drama.

    1. They already tried botox with limited results. They got some people with good growth though. Maybe botox and Propecia combo?

      1. “we envisage the development of the potential product candidate being completed by 2018.” -Fidia about Brotzu lotion.

        I’m not a fan really but I would buy it if it was available

  12. Every guy that believes the scalp tension theory is completely moronic. There have been loads of tests that prove it wrong, scalp hair that is transplanted to the arm falls out at the same rate as the remaining scalp hair. What about guys with DUPA? I know not everyone is a scientist here but just stop bringing up this false, bonehead theory

    1. Gary, what is your theory then?

      The article states that fibrosis, calcification, inflammation, etcetera are the results of a decades long tension process, especially at the top of your head. So if you do a hair transplant from the save donor area, it takes decades again before these fresh and strong follicles (the grafts also include surrounding healthy skin) have encountered the same tension, fibrosis, calcification, inflammation as those follicles that you have lost in that top area before.

      Some HT patients complain that after decades they do not only lose their last remaining native hair on top, but also the transplanted ones. This while they are not a high norwood, so the donor follicles should be lifelong growing tissue.

      (Your example) If you transplant follicles from the top to your arm, at the moment when you concluded you have AGA on top, then you know these transplanted ones including surrounding skin tissue (which already have suffered on the top for decades) will fail to stay on the recipient area (your arm). Those on your arm will go just like the native hair on your top, as the surrounding tissue (skin) already has been suffering.

      I won’t state this is the one-fits-all theory. However, I don’t think the authors are morons.

      1. Nedshed

        Look at children with progeria, who begin to bald in a male pattern at 3/4 years old. What about “decades of tension” then? What about galeatomy which has not helped a single balding person ever. I applaud people that come up with new theories but not if those theories are absolutely obliterated by available evidence. Fibrosis and subsequent calcification are common endpoints of inflammation of any kind, such as in atherosclerosis in the arteries or in MS where the myelin sheaths of axons are destroyed. It is absolutely not indicative of tension stress.

              1. “This relative microvascular insufficiency possibly results because frontal and vertex areas are supplied by supratrochlear and supraorbital arteries, which are smaller branches of internal carotid artery, and it overlies galea aponeurotica, which is relatively less vascular as compared to the temporal and occipital areas, which overlie muscles.[9] The androgenetic theory states that minituarization of hair follicle results from the action of DHT. Hair follicles contain 5α-reductase enzyme that is responsible for peripheral conversion of testosterone to more active DHT. There is an increased accumulation of DHT in the affected follicle and disturbed DHT to estradiol ratio due to relative microvascular insufficiency, which leads to minituarization of hair follicle.[10]”

                Disturbed DHT to estradiol ratio due to less oxygen …

                1. You clearly do not know how the scientific process works. Of course people generate hypotheses and try and publish them in journals titled “medical hypotheses” and that is exactly what they are, hypotheses of which a tiny fraction turns out to be true. Just because it is published doesn’t mean it’s true. Something can only be confirmed if there is significant work done on that hypothesis. These hugely questionable studies: open label (researchers know who had active ingredient), very, very small test group, do not prove anything. I know we can’t all be experts but don’t act like one if you have no clue what you are doing.

                  1. Well Gary, no one here knows exactly what the solution to AGA is exactly, otherwise this site would not be visited by many hair loss sufferers, would it?! If the experts solved it, we wouldn’t be here.

                    I for instance try to stay open-minded, and therefore I am curious what your ideas are. You talk about DUPA, progeria, MS patients, which have even more severe problems than AGA patients. There are other genes involved when it comes to these diseases.

                    I think senescence might play a large role in AGA, the fact that the articles about tension and botox don’t mention senescence, does not mean I will call them bs immediatedly. At the same time we all are well aware of the fact we shouldn’t believe everything, including published articles.

  13. Below is a link to a recent article about University of Southern Cal research regarding RCGD423. If you recall, RCGD423 is one of the 2 molecules used in the UCLA research to activate stem cells in the hair follicle to grow hair. In this article, the molecule was used to proliferate cartilage cells and a clinical trial is planned. Evidence that another research team believes this molecule to be safe or possibly that toxicity studies have already been completed, clearing the way for a clinical trial. At any rate, I view this parallel research as a positive development with respect to speeding the clinical trial path forward. I am probably a little more optimistic than most about this potential treatment due to it’s simplicity as a shortcut around all of the complicated regenerative challenges: https://newatlas.com/rcgd-423-arthritis-molecule/53327/

    1. Great find. The word “inflammation” shows up up yet again.

      From the dailytrojan article, a good quote:

      “In the lab, RCGD423 has already been proven effective to activate stem cells to make hair grow”

      Also, in my article:

      https://www.hairlosscure2020.com/category/uk5099/

      I mentioned that RCGD423, activates the JAK-STAT signaling pathway. Wonder if there is any strong link between RCGD423 and JAK inhibitors too?

      1. Good question! Although there are a number of researchers approaching this from different angles, there seems to be a fair degree of commonality. I don’t know much about getting a synthetic molecule approved for trial in humans, but I have read where they have to do a toxicity study in larger mammals. If they have already completed one, it is possible that we could have human clinical trial results by year end. I just saw a link stating that the use of RCGD423 to promote hair growth is covered by a provisional patent application filed by UC Regents and this technology has been licensed by a company called Carthronix LLC. See https://www.carthronix.com/pipeline

        1. Also see the technology page: https://www.carthronix.com/technology “Our studies have delineated the exact molecular mechanism of CX-1, positioning CarthroniX for rapid development and streamlined regulatory approval. In addition, the CX-1 molecule and the pathway it activates have potential roles in treatments for degenerative disc disease, wound healing and hair growth.” They talk in a bit more detail about the JAK/STAT pathway.

        2. Great find PinotQ. Too bad everything is still pre-clinical, but hopefully they can fast-track trials based on their website statements such as “rapid development and streamlined regulatory approval”.

          1. No telling what that means in terms of actual time line but I too was happy to see those statements. I checked the California Secretary of State’s web site and found that Carthronix (actually a Delaware Corp) was formed and registered in December of 2015. Hopefully that means they have some momentum already.

            1. FYI They apparently had a good idea of what they were looking for when they discovered/identfied RCGD 423. They used some type of technology called high throughput screening of 170,000 compounds to find a molecule that would do what they wanted it to do. I believe this is one of the reasons why they say on their website that they have delineated the exact molecular mechanism that should speed development and approval.

  14. If you guys really wanna get FIDIA’s attention and have them post some kinda news/information about the Brotzu lotion….just start one-starring their business page on google. In Google, search “Fidia Farmaceutici S.p.A. italy” and put a one star review, with a comment about Brotzu. I’m sure that would get their attention….and somehow get them to tell us what is going on with the Brotzu lotion.

      1. @That Guy….lol at you thinking something is petty and stupid. You have over 1000 posts on hair loss talk…most of which are pointless or criticize others/ideas. Your opinion is not needed…go back to hair loss talk and continue to pull hairs out of your ****

        1. Yeah, tell that to my like-to-post ratio and writing that was good enough to warrant actually writing FOR the site itself.

          The only time I criticize others’ ideas is when they’re stupid.

          Like your idea.

          Yes, let’s give a company 1 star ratings on internet! I’m sure they care deeply and that’ll show them for not divulging any potential trade secrets to random chrome domes online! Why don’t you go ahead and do that to Coca-Cola? Maybe they’ll give you the recipe!

          1. LOL…you write official stuff for a hair loss forum. You must be a big deal. My bad, I’ll back off. Have fun writing more stuff on hair loss forums and getting that high like to dislike ratio.

  15. Hey guys, I haven’t posted much on this site but I’ve been on it a lot from time to time reading the comments and articles and such. This is a little off topic, but I was wondering what some of you guys think about the PRP treatments? I think it definitely makes sense for it to work, but I was also wondering if injecting other things, such as liquid Niacin, would work. I know the science behind hair growth is a little trickier, but don’t you think it makes sense to inject something directly into the scalp?

    1. Hi Eric I totally agree with your theory. They are injecting dutasteride and fin into the scalp with some good regrowth. I also remember there was this Egyptian doctor in Egypt who had tremendous success with a minox cocktail injection that grew back tons of hair. This was back in 2007 but she was shut down. As for PRP I hear mix results. Some people get regrowth some lose more hair some people get nothing. There are a bunch of YouTube videos of people getting it done. I actually spoke to a few dermatologists avd some ht docs in the Philly area and they say they don’t offer PRP for hair due to its limited benefits. They say it works well for joint inflammation and skin issues.

    2. I have had PRP. Also tried the laser cap for a few months, plus vitamin supplements, scalp exercises, coconut oil, onions, NOFAP. They make no difference. Don’t waste your money or time.

      Regarding PRP, there was a lot of excitement about it a few years ago but the phrase they like to use now is “cosmetically insignificant”. For argument sake some scientist might show you a before and after picture at 100x magnification to prove a technical point, but if that is what it takes to tell the difference then what is the point?

      Not wanting to end on a negative tone, ride this year out and wait for the results of Aclaris and Replicel/Shiseido. The time will pass quickly. I don’t remember much since New Year and we are nearly half way through February!

  16. Awesome find Pinoq! Looks like aclaris has some competition on the jak front. Competition is our best friend fellas! I hope CX1 can pull through avd fast track in two years or better yet aclaris fast track quicker avd bring their jak to market in less time. This next 24 months will be make or break for us in terms of new treatments. I got money on follica, and aclaris for USA based treatments. Sisheido for Asian markets. If all fails Im putting my name on the wait list for that Madrid ht doctor coutos his work is bad ass! Maybe 3 years wait but a full head of hair will be worth the wait!

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